CausalSentinel

Protein Dossier — C10orf10 (Protein DEPP1)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Amyotrophic lateral sclerosis 0.0553 0.0224 0.0134 Wald ratio 1 trans NA
Squamous cell lung cancer -0.0918 0.0415 0.0272 Wald ratio 1 trans NA
Eczema 0.0504 0.0294 0.0865 Wald ratio 1 trans NA
Rheumatoid arthritis -0.0254 0.015 0.0899 Wald ratio 1 trans NA
Neuroticism 0.0086 0.00516 0.0956 Wald ratio 1 trans NA
High grade serous ovarian cancer -0.0257 0.0169 0.128 Wald ratio 1 trans NA
Lung cancer -0.042 0.0287 0.143 Wald ratio 1 trans NA
Caudate volume 12.4 8.66 0.152 Wald ratio 1 trans NA
Forearm bone mineral density -0.086 0.0791 0.277 Wald ratio 1 trans NA
Alzheimer’s disease -0.0183 0.0171 0.284 Wald ratio 1 trans NA
Femoral neck bone mineral density -0.0349 0.0337 0.301 Wald ratio 1 trans NA
Amygdala volume -3.98 4.23 0.346 Wald ratio 1 trans NA
…and 5 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

No GWAS Catalog associations mapped to this gene.

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 106 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
placental abruption 0.076 common-variant locus no MR -> candidate analysis
malignant renal pelvis neoplasm 0.073 common-variant locus no MR -> candidate analysis
psoriatic arthritis 0.062 common-variant locus no MR -> candidate analysis
stroke disorder 0.053 common-variant locus no MR -> candidate analysis
alcohol drinking 0.053 common-variant locus no MR -> candidate analysis
cellulitis 0.048 common-variant locus no MR -> candidate analysis
abscess 0.048 common-variant locus no MR -> candidate analysis
Hydrocephalus 0.046 common-variant locus no MR -> candidate analysis
prostate carcinoma 0.042 common-variant locus no MR -> candidate analysis

Of the 9 rows above, 9 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint not available
GWAS Catalog 1 unique SNPs / 2 rows
ClinVar no records
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance