CausalSentinel

Protein Dossier — C17orf78 (Uncharacterized protein C17orf78)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Non-cancer illness code self-reported: enlarged prostate 0.248 0.0733 7.33e-04 Wald ratio 1 trans NA
Non-cancer illness code self-reported: pneumothorax 0.772 0.269 0.00407 Wald ratio 1 trans NA
Non-cancer illness code self-reported: psoriasis 0.23 0.0823 0.00528 Wald ratio 1 trans NA
Fracture resulting from simple fall 0.0709 0.027 0.00859 Wald ratio 1 trans NA
Diagnoses - main ICD10: N40 Hyperplasia of prostate 0.214 0.0927 0.0211 Wald ratio 1 trans NA
Invasive mucinous ovarian cancer -0.489 0.212 0.0211 Wald ratio 1 trans NA
Diagnoses - main ICD10: R55 Syncope and collapse 0.214 0.0945 0.0238 Wald ratio 1 trans NA
Intracranial volume -2.07e+04 9.19e+03 0.0243 Wald ratio 1 trans NA
Pallidum volume -20.3 9.12 0.026 Wald ratio 1 trans NA
Hearing difficulty or problems: Yes 0.0394 0.0181 0.0298 Wald ratio 1 trans NA
Diagnoses - main ICD10: L03 Cellulitis -0.403 0.19 0.0338 Wald ratio 1 trans NA
Non-cancer illness code self-reported: hypertrophic cardiomyopathy (hcm or hocm) 0.807 0.411 0.0496 Wald ratio 1 trans NA
…and 65 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

1 association rows across 1 traits (0 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Retinal nerve fibre layer (RNFL) thickness 9e-7 rs34232224 1 GCST90554824 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 19 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
Abnormality of the skeletal system 0.232 common-variant locus no MR -> candidate analysis
Abnormality of the integument 0.146 common-variant locus no MR -> candidate analysis
insomnia 0.146 common-variant locus no MR -> candidate analysis
secondary malignant neoplasm 0.12 common-variant locus no MR -> candidate analysis
intracranial hemorrhage 0.096 common-variant locus no MR -> candidate analysis
alcohol drinking 0.059 common-variant locus no MR -> candidate analysis

Of the 6 rows above, 6 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=1.3e-08, LOEUF=1.17 — LoF-tolerant
GWAS Catalog 32 unique SNPs / 60 rows
ClinVar 158 records; 21 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance