CausalSentinel

Protein Dossier — C1QL1 (C1q-related factor)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Systolic blood pressure automated reading 0.0377 0.00883 1.91e-05 Wald ratio 1 cis NA
Diagnoses - main ICD10: K43 Ventral hernia 0.331 0.094 4.33e-04 Wald ratio 1 cis NA
Heel bone mineral density (BMD) T-score automated 0.0384 0.0112 5.81e-04 Wald ratio 1 cis NA
Forced vital capacity (FVC) -0.024 0.00708 6.96e-04 Wald ratio 1 cis NA
Height -0.0358 0.0108 8.58e-04 Wald ratio 1 cis NA
Fractured bone site(s): Wrist 0.168 0.0522 0.00126 Wald ratio 1 cis NA
Iron 0.106 0.0362 0.00338 Wald ratio 1 cis NA
Forced expiratory volume in 1-second (FEV1) -0.0201 0.00747 0.007 Wald ratio 1 cis NA
Transferrin Saturation 0.0914 0.0366 0.0124 Wald ratio 1 cis NA
Diastolic blood pressure automated reading 0.0202 0.00883 0.0223 Wald ratio 1 cis NA
Serum creatinine (eGFRcrea) 0.00717 0.0033 0.0297 Wald ratio 1 cis NA
Hirschsprung’s disease -1.03 0.48 0.0326 Wald ratio 1 cis NA
…and 104 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

4 association rows across 4 traits (2 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
C1q-related factor levels 6e-68 rs1055646 1 GCST90246765 no MR -> candidate analysis
Serum levels of protein C1QL1 3e-53 rs1055646 1 GCST90089401 no MR -> candidate analysis
DNA methylation (variation) 2e-6 rs1007190 1 GCST002058 no MR -> candidate analysis
Dry eye disease with Sjogren-like syndromes 4e-6 rs3024285 1 GCST90444403 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 115 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
anxiety disorder 0.31 common-variant locus no MR -> candidate analysis
epilepsy 0.164 common-variant locus no MR -> candidate analysis
enteritis 0.123 common-variant locus no MR -> candidate analysis
intelligence 0.112 common-variant locus no MR -> candidate analysis

Of the 4 rows above, 4 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=3.6e-06, LOEUF=1.62 — LoF-tolerant
GWAS Catalog 74 unique SNPs / 148 rows
ClinVar 56 records; 2 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance