CausalSentinel

Protein Dossier — C1QTNF5 (Complement C1q tumor necrosis factor-related protein 5)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Crohn’s disease -0.612 0.0489 7.15e-36 Wald ratio 1 trans 1.52e-05
Inflammatory bowel disease -0.445 0.0404 3.06e-28 Wald ratio 1 trans 1.54e-12
Ulcerative colitis -0.267 0.0509 1.50e-07 Wald ratio 1 trans 4.17e-14
Microalbuminuria 0.191 0.0562 6.70e-04 Inverse variance weighted 3 trans NA
Microalbuminuria 0.191 0.0562 6.70e-04 Inverse variance weighted 3 cis NA
Microalbuminuria 0.191 0.0562 6.70e-04 Inverse variance weighted 3 trans NA
Urinary albumin-to-creatinine ratio 0.051 0.0163 0.00174 Inverse variance weighted 3 trans NA
Urinary albumin-to-creatinine ratio 0.051 0.0163 0.00174 Inverse variance weighted 3 cis NA
Urinary albumin-to-creatinine ratio 0.051 0.0163 0.00174 Inverse variance weighted 3 trans NA
Triglycerides 0.0383 0.0133 0.00384 Inverse variance weighted 3 trans NA
Triglycerides 0.0383 0.0133 0.00384 Inverse variance weighted 3 cis NA
Triglycerides 0.0383 0.0133 0.00384 Inverse variance weighted 3 trans NA
…and 308 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

2 association rows across 2 traits (2 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
C1QTNF5 protein levels 4e-68 rs2509656 1 GCST90468489 no MR -> candidate analysis
Complement C1q tumor necrosis factor-related protein 5 level 4e-44 rs2509656 1 GCST90246768 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 967 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
late-onset retinal degeneration 0.789 established (curated) no MR -> candidate analysis
Retinal dystrophy 0.833 established (curated) no MR -> candidate analysis
retinitis pigmentosa 0.195 established (curated) no MR -> candidate analysis
isolated microphthalmia 5 0.313 established (curated) no MR -> candidate analysis
hereditary disease 0.307 established (curated) no MR -> candidate analysis
gout 0.268 common-variant locus no MR -> candidate analysis
nanophthalmos 2 0.195 established (curated) no MR -> candidate analysis

Of the 7 rows above, 7 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=0.19, LOEUF=0.966 — LoF-tolerant
GWAS Catalog 71 unique SNPs / 142 rows
ClinVar 963 records; 5 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance