CausalSentinel

Protein Dossier — C8orf33 (UPF0488 protein C8orf33)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Diagnoses - main ICD10: N20 Calculus of kidney and ureter 0.297 0.107 0.00535 Wald ratio 1 trans NA
Non-cancer illness code self-reported: pernicious anaemia 0.387 0.153 0.0117 Wald ratio 1 trans NA
Non-cancer illness code self-reported: gout -0.277 0.135 0.0398 Wald ratio 1 trans NA
Hirschsprung’s disease 1.48 0.723 0.04 Wald ratio 1 trans NA
Body fat 0.054 0.0266 0.042 Wald ratio 1 trans NA
Neo-neuroticism -0.903 0.462 0.0504 Wald ratio 1 trans NA
Neo-agreeableness 0.577 0.298 0.0526 Wald ratio 1 trans NA
Diagnoses - main ICD10: Z09 Follow-up examination after treatment for conditions other than malignant neoplasms 0.154 0.0837 0.0658 Wald ratio 1 trans NA
Lung cancer -0.166 0.0918 0.0701 Wald ratio 1 trans NA
Non-cancer illness code self-reported: hypertension -0.0372 0.0211 0.0774 Wald ratio 1 trans NA
Diagnoses - main ICD10: M72 Fibroblastic disorders 0.231 0.131 0.0786 Wald ratio 1 trans NA
Large vessel disease -0.29 0.169 0.0868 Wald ratio 1 trans NA
…and 98 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

No GWAS Catalog associations mapped to this gene.

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 29 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
ulcerative colitis 0.371 common-variant locus MR: beta=0.054, p=0.399 (trans)
placenta praevia 0.155 common-variant locus no MR -> candidate analysis
metabolic dysfunction-associated steatotic liver disease 0.037 common-variant locus no MR -> candidate analysis

Of the 3 rows above, 2 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=6.7e-11, LOEUF=1.49 — LoF-tolerant
GWAS Catalog 5 unique SNPs / 10 rows
ClinVar 75 records; 12 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance