CausalSentinel

Protein Dossier — DNAJC30 (DnaJ homolog subfamily C member 30, mitochondrial)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Eye problems or disorders: Glaucoma -0.28 0.0949 0.00321 Wald ratio 1 cis NA
Birth weight 0.0372 0.0131 0.00451 Wald ratio 1 cis NA
Eczema 0.193 0.071 0.00666 Wald ratio 1 cis NA
Diagnoses - main ICD10: Z09 Follow-up examination after treatment for conditions other than malignant neoplasms 0.15 0.0594 0.0114 Wald ratio 1 cis NA
Body fat -0.491 0.216 0.0231 Wald ratio 1 cis NA
Non-cancer illness code self-reported: anxiety or panic attacks 0.136 0.0639 0.0332 Wald ratio 1 cis NA
Lumbar spine bone mineral density 0.066 0.0318 0.0378 Wald ratio 1 cis NA
ER-positive Breast cancer (Combined Oncoarray; iCOGS; GWAS meta analysis) 0.0635 0.0306 0.0382 Wald ratio 1 cis NA
Non-cancer illness code self-reported: emphysema or chronic bronchitis 0.132 0.0644 0.0408 Wald ratio 1 cis NA
Diagnoses - main ICD10: R35 Polyuria 0.215 0.111 0.0527 Wald ratio 1 cis NA
Non-cancer illness code self-reported: iron deficiency anaemia -0.292 0.16 0.068 Wald ratio 1 cis NA
Primary sclerosing cholangitis -0.243 0.135 0.0715 Wald ratio 1 cis NA
…and 62 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

22 association rows across 18 traits (22 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
DnaJ homolog subfamily C member 30 levels 3e-155 rs113428756 1 GCST90427118 no MR -> candidate analysis
Triglyceride levels 9e-56 rs13242693 2 GCST90019523 no MR -> candidate analysis
Height 4e-45 rs8891 1 GCST90245848 no MR -> candidate analysis
Drinks per week 4e-19 rs13243804 1 GCST90243989 no MR -> candidate analysis
DnaJ homolog subfamily C member 30 level in Chronic kidney d 2e-17 rs113239638 1 GCST90238677 no MR -> candidate analysis
Polyunsaturated fatty acids to monounsaturated fatty acids r 9e-17 rs1128349 1 GCST90502566 no MR -> candidate analysis
Gamma glutamyl transferase levels 2e-16 rs13242693 1 GCST90019507 no MR -> candidate analysis
Low-density lipoprotein levels (MTAG) 7e-16 rs1128349 1 GCST90179148 no MR -> candidate analysis
Monounsaturated fatty acid levels 7e-16 rs13243804 2 GCST90502358 no MR -> candidate analysis
Omega-6 fatty acids to total fatty acids percentage 1e-13 rs8891 2 GCST90502528 no MR -> candidate analysis
Aspartate aminotransferase to alanine aminotransferase ratio 1e-12 rs13242693 1 GCST90019498 no MR -> candidate analysis
Polyunsaturated fatty acids to total fatty acids percentage 3e-11 rs8891 2 GCST90502632 no MR -> candidate analysis
…and 6 more traits (see JSON)          

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 90 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
Leber-like hereditary optic neuropathy, autosomal recessive 1 0.853 established (curated) no MR -> candidate analysis
Leber hereditary optic neuropathy 0.598 established (curated) no MR -> candidate analysis
Leber hereditary optic neuropathy, autosomal recessive 0.687 established (curated) no MR -> candidate analysis
optic atrophy 0.666 established (curated) no MR -> candidate analysis
Retinal dystrophy 0.605 established (curated) no MR -> candidate analysis
Abnormality of the skeletal system 0.082 common-variant locus no MR -> candidate analysis
gout 0.082 common-variant locus no MR -> candidate analysis
type 2 diabetes mellitus 0.082 common-variant locus no MR -> candidate analysis
diabetes mellitus 0.046 common-variant locus no MR -> candidate analysis

Of the 9 rows above, 9 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=1.9e-06, LOEUF=1.58 — LoF-tolerant
GWAS Catalog 144 unique SNPs / 338 rows
ClinVar 225 records; 9 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance