CausalSentinel

Protein Dossier — ERMAP (Erythroid membrane-associated protein)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Diagnoses - main ICD10: I48 Atrial fibrillation and flutter 0.233 0.0971 0.0164 Wald ratio 1 trans NA
Non-cancer illness code self-reported: enlarged prostate 0.214 0.0893 0.0167 Wald ratio 1 trans NA
Diagnoses - main ICD10: K20 Oesophagitis 0.236 0.103 0.0225 Wald ratio 1 trans NA
Diagnoses - main ICD10: Z09 Follow-up examination after treatment for conditions other than malignant neoplasms -0.337 0.15 0.0248 Wald ratio 1 trans NA
Diagnoses - main ICD10: N40 Hyperplasia of prostate 0.233 0.107 0.0296 Wald ratio 1 trans NA
Fractured bone site(s): Wrist -0.252 0.12 0.0354 Wald ratio 1 trans NA
Fractured bone site(s): Arm 0.216 0.105 0.04 Wald ratio 1 trans NA
Diagnoses - main ICD10: R04 Haemorrhage from respiratory passages 0.281 0.138 0.0412 Wald ratio 1 trans NA
Pallidum volume -19.7 9.96 0.0485 Wald ratio 1 trans NA
Thalamus volume -61.6 32.9 0.0609 Wald ratio 1 trans NA
Non-cancer illness code self-reported: asthma -0.0718 0.0386 0.0631 Wald ratio 1 trans NA
Non-cancer illness code self-reported: kidney stone or ureter stone or bladder stone 0.219 0.118 0.0645 Wald ratio 1 trans NA
…and 69 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

6 association rows across 5 traits (4 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
ERMAP protein levels 1e-36 rs3214967 1 GCST90469147 no MR -> candidate analysis
Red blood cell erythrocyte distribution width (UKB data fiel 1e-13 rs34441268 1 GCST90468099 no MR -> candidate analysis
Red cell distribution width 5e-12 rs12406643 2 GCST007074 no MR -> candidate analysis
Stuttering 2e-6 rs567238919 1 GCST90707226 no MR -> candidate analysis
Visceral fat 6e-6 rs72666872 1 GCST008473 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 81 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
hypertensive disorder 0.474 common-variant locus no MR -> candidate analysis
cardiovascular disorder 0.356 common-variant locus no MR -> candidate analysis
placental abruption 0.045 common-variant locus no MR -> candidate analysis

Of the 3 rows above, 3 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=2.1e-08, LOEUF=0.915 — LoF-tolerant
GWAS Catalog 23 unique SNPs / 46 rows
ClinVar 103 records; 0 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance