CausalSentinel

Protein Dossier — FLRT3 (Leucine-rich repeat transmembrane protein FLRT3)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Femoral neck bone mineral density 0.078 0.0197 7.57e-05 Wald ratio 1 cis NA
ER-negative Breast cancer (Combined Oncoarray; iCOGS; GWAS meta analysis) -0.11 0.0315 4.78e-04 Wald ratio 1 cis NA
Diastolic blood pressure automated reading -0.0213 0.00643 9.25e-04 Wald ratio 1 cis NA
Heel bone mineral density (BMD) T-score automated 0.0247 0.00811 0.0023 Wald ratio 1 cis NA
Lumbar spine bone mineral density 0.0678 0.0231 0.00329 Wald ratio 1 cis NA
Breast cancer (Combined Oncoarray; iCOGS; GWAS meta analysis) -0.0475 0.0171 0.0055 Wald ratio 1 cis NA
Fractured bone site(s): Wrist -0.13 0.0511 0.011 Wald ratio 1 cis NA
Fractured or broken bones in last 5 years -0.0509 0.0206 0.0135 Wald ratio 1 cis NA
Intracranial volume -1.21e+04 4.92e+03 0.0136 Wald ratio 1 cis NA
Non-cancer illness code self-reported: enlarged prostate -0.154 0.0628 0.0144 Wald ratio 1 cis NA
Cough on most days -0.084 0.035 0.0163 Wald ratio 1 cis NA
Non-cancer illness code self-reported: gout 0.112 0.0471 0.0171 Wald ratio 1 cis NA
…and 73 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

1 association rows across 1 traits (0 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Mood disorder in prion disease 7e-6 rs761998 1 GCST002864 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 140 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
Kallmann syndrome 0.814 established (curated) no MR -> candidate analysis
hypogonadotropic hypogonadism 21 with or without anosmia 0.696 established (curated) no MR -> candidate analysis
bipolar disorder 0.565 common-variant locus MR: beta=0.149, p=0.166 (cis)
alcohol drinking 0.241 common-variant locus no MR -> candidate analysis
attention deficit-hyperactivity disorder 0.21 common-variant locus no MR -> candidate analysis
intelligence 0.211 common-variant locus no MR -> candidate analysis
smoking initiation 0.21 common-variant locus no MR -> candidate analysis
adverse effect 0.203 common-variant locus no MR -> candidate analysis
response to stimulus 0.203 common-variant locus no MR -> candidate analysis
pathological myopia 0.182 established (curated) no MR -> candidate analysis
Genetic 46,XY disorder of sex development 0.182 established (curated) no MR -> candidate analysis
myopia 0.182 established (curated) no MR -> candidate analysis
amenorrhea 0.182 established (curated) no MR -> candidate analysis
disorder of sexual differentiation 0.182 established (curated) no MR -> candidate analysis
chronic venous hypertension 0.172 common-variant locus no MR -> candidate analysis

Of the 15 rows above, 14 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=0.44, LOEUF=0.599 — LoF-tolerant
GWAS Catalog 8 unique SNPs / 16 rows
ClinVar 150 records; 0 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance