CausalSentinel

Protein Dossier — GAL (Germinal center-associated signaling and motility protein)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Glioma -0.382 0.248 0.124 Wald ratio 1 trans NA

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

53 association rows across 34 traits (49 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Bone mineral density mean 7e-77 rs186637849 6 GCST90321120 no MR -> candidate analysis
Circulating GAL levels 1e-55 rs2012605 3 GCST90860395 no MR -> candidate analysis
Appendicular lean mass 7e-53 rs7129320 2 GCST90000025 no MR -> candidate analysis
GAL protein levels 2e-50 rs3018721 2 GCST90469300 no MR -> candidate analysis
Whole body water mass (UKB data field 23102) 2e-44 rs7129320 1 GCST90468184 no MR -> candidate analysis
Fracture 8e-44 rs35989399 1 GCST006980 no MR -> candidate analysis
Height 1e-43 rs2510396 5 GCST007841 no MR -> candidate analysis
Height (baseline) 3e-41 rs7102308 2 GCST90565843 no MR -> candidate analysis
Basal metabolic rate (UKB data field 23105) 4e-39 rs7129320 1 GCST90468159 no MR -> candidate analysis
Heel bone mineral density 3e-38 rs2510405 1 GCST006433 no MR -> candidate analysis
Ximenoylcarnitine (C26:1) levels 2e-36 rs2510374 1 GCST90200149 no MR -> candidate analysis
Cerotoylcarnitine (C26) levels 2e-34 rs112549564 3 GCST90200136 no MR -> candidate analysis
…and 22 more traits (see JSON)          

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 1029 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
temporal lobe epilepsy 0.608 established (curated) no MR -> candidate analysis
autosomal dominant epilepsy with auditory features 0.674 established (curated) no MR -> candidate analysis
ankylosing spondylitis 0.448 common-variant locus no MR -> candidate analysis
Abnormality of the skeletal system 0.189 common-variant locus no MR -> candidate analysis
arthropathy 0.169 common-variant locus no MR -> candidate analysis
non-autoimmune hemolytic anemia 0.169 common-variant locus no MR -> candidate analysis
bone fracture 0.138 common-variant locus no MR -> candidate analysis
hair color 0.122 common-variant locus no MR -> candidate analysis

Of the 8 rows above, 8 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability 0 known modulators (Galectin-9C)
gnomAD constraint pLI=3.5e-05, LOEUF=1.39 — LoF-tolerant
GWAS Catalog 132 unique SNPs / 208 rows
ClinVar 90 records; 4 pathogenic in sample of 30
PharmGKB/ClinPGx 3 clinical annotations across 6 drugs

Caveats declared by the tools

Sources

Provenance