CausalSentinel

Protein Dossier — IGFLR1 (IGF-like family receptor 1)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Body fat 0.036 0.0116 0.00186 Wald ratio 1 cis NA
Major depressive disorder 0.122 0.0421 0.00385 Wald ratio 1 cis NA
Knee and hip osteoarthritis -0.123 0.0437 0.00486 Wald ratio 1 cis NA
Years of schooling -0.0186 0.00714 0.00932 Wald ratio 1 cis NA
Systolic blood pressure automated reading 0.0124 0.00499 0.0127 Wald ratio 1 cis NA
Diagnoses - main ICD10: C50 Malignant neoplasm of breast -0.105 0.0425 0.0136 Wald ratio 1 cis NA
Diastolic blood pressure automated reading 0.012 0.00499 0.0163 Wald ratio 1 cis NA
Diagnoses - main ICD10: G47 Sleep disorders -0.174 0.0763 0.0223 Wald ratio 1 cis NA
Body mass index (BMI) 0.0106 0.00488 0.0299 Wald ratio 1 cis NA
Childhood intelligence -0.0541 0.0263 0.0394 Wald ratio 1 cis NA
Haemoglobin concentration 0.0277 0.0136 0.0411 Wald ratio 1 cis NA
Hearing difficulty or problems: Yes -0.0173 0.00858 0.0434 Wald ratio 1 cis NA
…and 101 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

5 association rows across 4 traits (5 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
IGF-like family receptor 1 levels 1e-278 rs2871921 2 GCST90248013 no MR -> candidate analysis
IGF-like family receptor 1 levels (IGFLR1.7244.16.3) 6e-100 rs12459634 1 GCST90241467 no MR -> candidate analysis
Red cell distribution width 1e-15 rs12459634 1 GCST90002404 no MR -> candidate analysis
Lichen sclerosus 1e-10 rs140952221 1 GCST90824102 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 57 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
lichen sclerosus et atrophicus 0.614 common-variant locus no MR -> candidate analysis
dermatitis 0.127 common-variant locus no MR -> candidate analysis
Eczematoid dermatitis 0.127 common-variant locus no MR -> candidate analysis
spinal cord injury 0.068 common-variant locus no MR -> candidate analysis
mathematical ability 0.041 common-variant locus no MR -> candidate analysis

Of the 5 rows above, 5 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability 0 known modulators (IGF-like family receptor 1)
gnomAD constraint pLI=4.8e-05, LOEUF=1.09 — LoF-tolerant
GWAS Catalog 72 unique SNPs / 144 rows
ClinVar 95 records; 1 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance