CausalSentinel

Protein Dossier — NUDT16L1 (Tudor-interacting repair regulator protein)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Diagnoses - main ICD10: N81 Female genital prolapse -0.185 0.0786 0.0186 Wald ratio 1 trans NA
Fasting proinsulin -0.0765 0.0353 0.0303 Wald ratio 1 trans NA
Urate 0.0618 0.0288 0.0321 Wald ratio 1 trans NA
Diagnoses - main ICD10: R55 Syncope and collapse -0.216 0.104 0.0367 Wald ratio 1 trans NA
Alcohol intake frequency -0.0223 0.0115 0.0517 Wald ratio 1 trans NA
LDL cholesterol 0.0491 0.0253 0.0522 Wald ratio 1 trans NA
Hippocampus volume 31.4 16.2 0.0523 Wald ratio 1 trans NA
Cancer code self-reported: prostate cancer 0.149 0.0789 0.0585 Wald ratio 1 trans NA
Forced vital capacity (FVC) 0.012 0.00637 0.0598 Wald ratio 1 trans NA
Total cholesterol 0.0465 0.0247 0.06 Wald ratio 1 trans NA
Non-cancer illness code self-reported: ankylosing spondylitis 0.224 0.119 0.0608 Wald ratio 1 trans NA
Diagnoses - main ICD10: S66 Injury of muscle and tendon at wrist and hand level 0.28 0.151 0.0635 Wald ratio 1 trans NA
…and 80 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

5 association rows across 4 traits (5 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Height 1e-89 rs841217 2 GCST90245848 no MR -> candidate analysis
Physical function (baseline) 3e-15 rs841217 1 GCST90565837 no MR -> candidate analysis
Body size (confirmatory factor analysis Factor 21) 3e-12 rs841217 1 GCST90309355 no MR -> candidate analysis
Gamma glutamyl transferase levels 5e-10 rs841219 1 GCST90428730 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 65 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
systemic inflammatory response syndrome 0.049 common-variant locus no MR -> candidate analysis
type 2 diabetes mellitus 0.036 common-variant locus no MR -> candidate analysis
diabetes mellitus 0.036 common-variant locus no MR -> candidate analysis

Of the 3 rows above, 3 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability 0 known modulators (Tudor-interacting repair regulator protein)
gnomAD constraint pLI=9.6e-08, LOEUF=1.61 — LoF-tolerant
GWAS Catalog 60 unique SNPs / 120 rows
ClinVar 93 records; 0 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance