CausalSentinel

Protein Dossier — POFUT2 (GDP-fucose protein O-fucosyltransferase 2)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Diagnoses - main ICD10: K80 Cholelithiasis 0.0825 0.0264 0.00179 Wald ratio 1 trans NA
Body mass index (BMI) 0.0128 0.00412 0.00196 Wald ratio 1 trans NA
Diagnoses - main ICD10: I80 Phlebitis and thrombophlebitis 0.134 0.0529 0.011 Wald ratio 1 trans NA
Diagnoses - main ICD10: N20 Calculus of kidney and ureter 0.107 0.0442 0.0155 Wald ratio 1 trans NA
Diagnoses - main ICD10: R04 Haemorrhage from respiratory passages 0.107 0.0516 0.0374 Wald ratio 1 trans NA
Non-cancer illness code self-reported: osteoarthritis -0.029 0.0142 0.0413 Wald ratio 1 trans NA
Diagnoses - main ICD10: L03 Cellulitis 0.0847 0.0422 0.0446 Wald ratio 1 trans NA
Pulse rate 0.0145 0.00727 0.0459 Wald ratio 1 trans NA
Diagnoses - main ICD10: M72 Fibroblastic disorders 0.0982 0.0509 0.0539 Wald ratio 1 trans NA
Eye problems or disorders: Diabetes related eye disease 0.0927 0.0482 0.0543 Wald ratio 1 trans NA
Fracture resulting from simple fall 0.0205 0.0106 0.0544 Wald ratio 1 trans NA
Non-cancer illness code self-reported: iron deficiency anaemia -0.122 0.0641 0.0574 Wald ratio 1 trans NA
…and 53 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

2 association rows across 2 traits (0 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Suicide attempt severity in mood disorders 2e-6 rs74961332 1 GCST012279 no MR -> candidate analysis
Coronary artery calcified atherosclerotic plaque (90 or 130 8e-6 rs4819052 1 GCST005175 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 163 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
cataract 0.501 common-variant locus no MR -> candidate analysis
alcohol drinking 0.308 common-variant locus no MR -> candidate analysis
metabolic syndrome 0.134 common-variant locus no MR -> candidate analysis
major depressive disorder 0.125 common-variant locus no MR -> candidate analysis
type 2 diabetes mellitus 0.117 common-variant locus no MR -> candidate analysis
attention deficit-hyperactivity disorder 0.117 common-variant locus no MR -> candidate analysis

Of the 6 rows above, 6 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=4.5e-05, LOEUF=0.74 — LoF-tolerant
GWAS Catalog 36 unique SNPs / 72 rows
ClinVar 190 records; 1 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance