CausalSentinel

Protein Dossier — PSG3 (Pregnancy-specific beta-1-glycoprotein 3)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Clear cell ovarian cancer -0.209 0.0663 0.00163 Wald ratio 1 cis NA
Femoral neck bone mineral density -0.0234 0.0122 0.0555 Wald ratio 1 cis NA
Eczema 0.056 0.0296 0.0583 Wald ratio 1 cis NA
Rheumatoid arthritis 0.0601 0.0376 0.11 Wald ratio 1 cis NA
Bulimia nervosa 0.0276 0.0184 0.134 Wald ratio 1 cis NA
Forearm bone mineral density 0.0349 0.0241 0.148 Wald ratio 1 cis NA
Intracranial volume -3.98e+03 3.11e+03 0.2 Wald ratio 1 cis NA
Depressive symptoms -0.00767 0.00613 0.211 Wald ratio 1 cis NA
Myocardial infarction -0.0208 0.0167 0.211 Wald ratio 1 cis NA
Coronary heart disease -0.0185 0.0154 0.228 Wald ratio 1 cis NA
Thalamus volume -12 10 0.231 Wald ratio 1 cis NA
Lung cancer -0.0339 0.0289 0.242 Wald ratio 1 cis NA
…and 9 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

8 association rows across 5 traits (8 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
CEACAM1 protein levels 3e-32 rs147492478 1 GCST90468692 no MR -> candidate analysis
Low density lipoprotein cholesterol levels 2e-17 rs538657595 2 GCST90239658 no MR -> candidate analysis
PSG1 protein levels 1e-16 rs141072282 1 GCST90470353 no MR -> candidate analysis
Non-HDL cholesterol levels 3e-11 rs538657595 2 GCST90239670 no MR -> candidate analysis
Total cholesterol levels 1e-10 rs538657595 2 GCST90239676 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 75 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
intestinal impaction 0.416 common-variant locus no MR -> candidate analysis
venous thromboembolism 0.093 common-variant locus no MR -> candidate analysis
Alzheimer disease 0.055 common-variant locus no MR -> candidate analysis

Of the 3 rows above, 3 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=8.2e-26, LOEUF=1.59 — LoF-tolerant
GWAS Catalog 22 unique SNPs / 44 rows
ClinVar 162 records; 1 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance