CausalSentinel

Protein Dossier — RDH16 (Retinol dehydrogenase 16)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Cancer code self-reported: prostate cancer 0.29 0.127 0.0225 Wald ratio 1 trans NA

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

29 association rows across 18 traits (21 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Circulating ERBB3 levels 2e-85 rs2629411 1 GCST90860027 no MR -> candidate analysis
Circulating NOTCH3 levels 7e-36 rs2279373 1 GCST90859932 no MR -> candidate analysis
Headache or migraine 2e-21 rs4759042 2 GCST90267554 no MR -> candidate analysis
Mean platelet thrombocyte volume (UKB data field 30100) 6e-20 rs28470625 1 GCST90468087 no MR -> candidate analysis
Circulating EBI3_IL27 levels 2e-19 rs2279373 2 GCST90859764 no MR -> candidate analysis
Free androgen index 4e-17 rs34138930 1 GCST90239823 no MR -> candidate analysis
Free testosterone levels 2e-16 rs34138930 2 GCST90239825 no MR -> candidate analysis
Bioavailable testosterone levels 4e-15 rs34138930 3 GCST90012103 no MR -> candidate analysis
Insomnia 2e-12 rs1098740 3 GCST90131901 no MR -> candidate analysis
Heel bone mineral density 1e-11 rs2279743 4 GCST006288 no MR -> candidate analysis
Respiratory diseases 2e-9 rs10506346 1 GCST007076 no MR -> candidate analysis
Gut microbial network clusters (Pink (at 1 year) x Any Breas 1e-8 rs61939617 1 GCST90569309 no MR -> candidate analysis
…and 6 more traits (see JSON)          

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 105 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
asthma 0.282 common-variant locus no MR -> candidate analysis

Of the 1 rows above, 1 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=5.2e-10, LOEUF=1.56 — LoF-tolerant
GWAS Catalog 65 unique SNPs / 130 rows
ClinVar 83 records; 1 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance