CausalSentinel

Protein Dossier — S100A5 (Protein S100-A5)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Low grade serous ovarian cancer -0.394 0.134 0.00325 Wald ratio 1 trans NA
Non-cancer illness code self-reported: hiatus hernia 0.106 0.0372 0.00432 Wald ratio 1 trans NA
Rheumatoid arthritis 0.107 0.0377 0.0044 Wald ratio 1 trans NA
Diagnoses - main ICD10: K29 Gastritis and duodenitis -0.124 0.0462 0.00709 Wald ratio 1 trans NA
Non-cancer illness code self-reported: hypertrophic cardiomyopathy (hcm or hocm) 0.64 0.25 0.0104 Wald ratio 1 trans NA
Forearm bone mineral density -0.0995 0.0397 0.0123 Wald ratio 1 trans NA
Eye problems or disorders: Glaucoma -0.15 0.0612 0.0141 Wald ratio 1 trans NA
Platelet count 2.42 1.07 0.0241 Wald ratio 1 trans NA
Anorexia nervosa 0.175 0.0809 0.0303 Wald ratio 1 trans NA
Fasting glucose 0.0173 0.00815 0.0334 Wald ratio 1 trans NA
Diagnoses - main ICD10: N20 Calculus of kidney and ureter -0.194 0.0917 0.0348 Wald ratio 1 trans NA
Non-cancer illness code self-reported: muscle or soft tissue injuries 0.137 0.065 0.0357 Wald ratio 1 trans NA
…and 98 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

3 association rows across 3 traits (2 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Hematological traits (multi-trait analysis) 8e-29 rs1810765 1 GCST90838667 no MR -> candidate analysis
Atopic dermatitis 3e-8 rs141484567 1 GCST90244002 no MR -> candidate analysis
White blood cell count (monocyte) 5e-8 rs1810765 1 GCST90026507 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 59 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
liver disorder 0.066 common-variant locus no MR -> candidate analysis
osteomyelitis 0.055 common-variant locus no MR -> candidate analysis

Of the 2 rows above, 2 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability 0 known modulators (Protein S100-A5)
gnomAD constraint pLI=0.026, LOEUF=1.57 — LoF-tolerant
GWAS Catalog 54 unique SNPs / 106 rows
ClinVar 32 records; 8 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance