CausalSentinel

Protein Dossier — SEPT10 (Septin-10)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Childhood intelligence -0.112 0.0368 0.00239 Wald ratio 1 trans NA
Non-cancer illness code self-reported: hypopituitarism 0.579 0.223 0.0095 Wald ratio 1 trans NA
Body mass index (BMI) 0.0178 0.00691 0.00982 Wald ratio 1 trans NA
Diagnoses - main ICD10: R11 Nausea and vomiting 0.224 0.0887 0.0115 Wald ratio 1 trans NA
Knee osteoarthritis 0.208 0.0827 0.0119 Wald ratio 1 trans NA
Diagnoses - main ICD10: I48 Atrial fibrillation and flutter -0.203 0.0808 0.0121 Wald ratio 1 trans NA
Age at menarche -0.0406 0.0165 0.014 Wald ratio 1 trans NA
Ischemic stroke 0.109 0.0476 0.022 Wald ratio 1 trans NA
Sodium in urine 0.0154 0.0068 0.024 Wald ratio 1 trans NA
Microalbuminuria 0.127 0.0595 0.0326 Wald ratio 1 trans NA
Creatinine (enzymatic) in urine 0.0141 0.00662 0.0331 Wald ratio 1 trans NA
Eye problems or disorders: Glaucoma 0.108 0.052 0.0386 Wald ratio 1 trans NA
…and 109 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

No GWAS Catalog associations mapped to this gene.

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 56 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
nutritional deficiency disease 0.484 common-variant locus no MR -> candidate analysis
ocular hypotension 0.265 common-variant locus no MR -> candidate analysis
stroke disorder 0.262 common-variant locus no MR -> candidate analysis
alcohol drinking 0.262 common-variant locus no MR -> candidate analysis
placental abruption 0.099 common-variant locus no MR -> candidate analysis
lung abscess 0.094 common-variant locus no MR -> candidate analysis
bronchopneumonia 0.094 common-variant locus no MR -> candidate analysis
esophageal ulcer 0.051 common-variant locus no MR -> candidate analysis

Of the 8 rows above, 8 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint not available
GWAS Catalog 1 unique SNPs / 2 rows
ClinVar no records
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance