CausalSentinel

Protein Dossier — STX10 (Syntaxin-10)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Body mass index (BMI) 0.0128 0.00497 0.00996 Wald ratio 1 trans NA
Lung cancer -0.0869 0.0354 0.0139 Wald ratio 1 trans NA
Diagnoses - main ICD10: R04 Haemorrhage from respiratory passages 0.14 0.0603 0.0206 Wald ratio 1 trans NA
Underlying (primary) cause of death: ICD10: E85.4 Organ-limited amyloidosis 1.08 0.481 0.0247 Wald ratio 1 trans NA
Non-cancer illness code self-reported: joint disorder -0.186 0.0882 0.0349 Wald ratio 1 trans NA
Diagnoses - main ICD10: K20 Oesophagitis 0.0953 0.0457 0.0371 Wald ratio 1 trans NA
Non-cancer illness code self-reported: bone disorder 0.185 0.0889 0.0377 Wald ratio 1 trans NA
Diagnoses - main ICD10: J33 Nasal polyp 0.131 0.0636 0.0387 Wald ratio 1 trans NA
Non-cancer illness code self-reported: kidney stone or ureter stone or bladder stone 0.103 0.0508 0.043 Wald ratio 1 trans NA
Age at menopause -0.0751 0.0375 0.0455 Wald ratio 1 trans NA
Weight 0.00855 0.00439 0.0514 Wald ratio 1 trans NA
Diagnoses - main ICD10: M17 Gonarthrosis [arthrosis of knee] 0.0653 0.0336 0.0517 Wald ratio 1 trans NA
…and 103 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

3 association rows across 3 traits (2 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Venous thromboembolism 2e-9 rs7508633 1 GCST009030 no MR -> candidate analysis
Household income (MTAG) 3e-8 rs116712274 1 GCST009524 no MR -> candidate analysis
Triamcinolone acetonide-induced ocular hypertension 6e-7 rs546725617 1 GCST90244659 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 27 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
osteoarthritis, knee 0.441 common-variant locus MR: beta=-0.0372, p=0.377 (trans)
venous thromboembolism 0.409 common-variant locus no MR -> candidate analysis
brain aneurysm 0.197 common-variant locus no MR -> candidate analysis
skin neoplasm 0.197 common-variant locus no MR -> candidate analysis
exfoliation syndrome 0.079 common-variant locus no MR -> candidate analysis
Abnormality of the cardiovascular system 0.048 common-variant locus no MR -> candidate analysis
placental retention 0.043 common-variant locus no MR -> candidate analysis

Of the 7 rows above, 6 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=8.4e-13, LOEUF=1.36 — LoF-tolerant
GWAS Catalog 66 unique SNPs / 132 rows
ClinVar 86 records; 0 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance