CausalSentinel

Protein Dossier — SWAP70 (Switch-associated protein 70)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Non-cancer illness code self-reported: hypertension -0.108 0.018 1.67e-09 Wald ratio 1 cis 0.993
Diastolic blood pressure automated reading -0.0556 0.00967 9.34e-09 Wald ratio 1 cis 9.13e-05
Systolic blood pressure automated reading -0.0531 0.00967 4.01e-08 Wald ratio 1 cis 1.82e-06
Coronary heart disease -0.177 0.0363 1.18e-06 Wald ratio 1 cis NA
HbA1C 0.0542 0.0135 6.33e-05 Wald ratio 1 cis NA
Breast cancer (Combined Oncoarray; iCOGS; GWAS meta analysis) 0.0812 0.0244 8.90e-04 Wald ratio 1 cis NA
Pulse rate 0.0526 0.0166 0.00157 Wald ratio 1 cis NA
Myocardial infarction -0.125 0.0404 0.00195 Wald ratio 1 cis NA
Diagnoses - main ICD10: N81 Female genital prolapse 0.197 0.0651 0.00245 Wald ratio 1 cis NA
Non-cancer illness code self-reported: hypothyroidism or myxoedema -0.147 0.0488 0.00256 Wald ratio 1 cis NA
Chronic kidney disease -0.181 0.0602 0.0027 Wald ratio 1 cis NA
Diagnoses - main ICD10: I83 Varicose veins of lower extremities 0.165 0.0561 0.00336 Wald ratio 1 cis NA
…and 95 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

167 association rows across 93 traits (155 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Switch-associated protein 70 levels 3e-733 rs6483656 3 GCST90249745 no MR -> candidate analysis
Hematological traits (multi-trait analysis) 4e-80 rs10770059 3 GCST90838669 no MR -> candidate analysis
Mean spheric corpuscular volume 9e-66 rs415895 1 GCST90002397 no MR -> candidate analysis
Glycated haemoglobin HbA1c levels (UKB data field 30750) 1e-55 rs415895 1 GCST90468072 no MR -> candidate analysis
Serum levels of protein SWAP70 1e-53 rs360153 1 GCST90087522 no MR -> candidate analysis
Systolic blood pressure 2e-49 rs360153 14 GCST90662908 MR: beta=-0.0531, p=4.01e-08 (cis)
Mean sphered cell volume (UKB data field 30270) 4e-48 rs10770059 1 GCST90468089 no MR -> candidate analysis
Hemoglobin A1c levels 1e-45 rs4910498 1 GCST90018958 no MR -> candidate analysis
Platelet distribution width (UKB data field 30110) 3e-45 rs415895 1 GCST90468097 no MR -> candidate analysis
Diastolic blood pressure 6e-45 rs360153 13 GCST90662909 MR: beta=-0.0556, p=9.34e-09 (cis)
Mean corpuscular hemoglobin concentration 9e-41 rs415895 4 GCST90002391 no MR -> candidate analysis
Hemoglobin A1c (HbA1c, minimum, inv-norm transformed) 1e-37 rs415895 1 GCST90475097 no MR -> candidate analysis
…and 81 more traits (see JSON)          

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 127 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
hypertensive disorder 0.93 common-variant locus no MR -> candidate analysis
essential hypertension 0.846 common-variant locus no MR -> candidate analysis
cardiovascular disorder 0.774 common-variant locus no MR -> candidate analysis
coronary artery disorder 0.728 common-variant locus no MR -> candidate analysis
Increased blood pressure 0.718 common-variant locus no MR -> candidate analysis
Abnormality of the skeletal system 0.69 common-variant locus no MR -> candidate analysis
rheumatoid arthritis 0.631 common-variant locus no MR -> candidate analysis
hypothyroidism 0.633 common-variant locus MR: beta=-0.147, p=0.00256 (cis)
ischemic stroke 0.629 common-variant locus MR: beta=-0.135, p=0.0369 (cis)
heart failure 0.592 common-variant locus no MR -> candidate analysis
autoimmune disease 0.515 common-variant locus no MR -> candidate analysis
obesity disorder 0.512 common-variant locus no MR -> candidate analysis
alcohol drinking 0.504 common-variant locus no MR -> candidate analysis
urolithiasis 0.504 common-variant locus no MR -> candidate analysis
breast carcinoma 0.455 common-variant locus no MR -> candidate analysis

Of the 15 rows above, 13 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=0.087, LOEUF=0.564 — LoF-tolerant
GWAS Catalog 84 unique SNPs / 168 rows
ClinVar 75 records; 0 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance