CausalSentinel

Protein Dossier — TDGF1 (Protein Cripto)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Non-cancer illness code self-reported: gout 0.0524 0.017 0.00206 Wald ratio 1 cis NA
Nucleus accumbens volume -2.47 0.942 0.00864 Wald ratio 1 cis NA
Diagnoses - main ICD10: R14 Flatulence and related conditions 0.189 0.074 0.0109 Wald ratio 1 cis NA
Bulimia nervosa 0.0151 0.00605 0.0124 Wald ratio 1 cis NA
Heel bone mineral density (BMD) T-score automated -0.00693 0.00277 0.0125 Wald ratio 1 cis NA
Diagnoses - main ICD10: J33 Nasal polyp -0.0819 0.0337 0.0151 Wald ratio 1 cis NA
Microalbuminuria 0.0409 0.0174 0.0189 Wald ratio 1 cis NA
Diagnoses - main ICD10: C61 Malignant neoplasm of prostate -0.0657 0.028 0.019 Wald ratio 1 cis NA
Cancer code self-reported: prostate cancer -0.0594 0.0266 0.0253 Wald ratio 1 cis NA
Neuroticism 0.00605 0.00303 0.0455 Wald ratio 1 cis NA
Non-cancer illness code self-reported: gastro-oesophageal reflux (gord) or gastric reflux 0.0197 0.0101 0.0505 Wald ratio 1 cis NA
Non-cancer illness code self-reported: osteoarthritis -0.0139 0.00725 0.0558 Wald ratio 1 cis NA
…and 93 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

Dataset Trait Author Year
prot-c-2672_60_1 Cripto Suhre K 2019

3. GWAS Catalog results — traits with signal at this locus

No GWAS Catalog associations mapped to this gene.

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 280 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
midline interhemispheric variant of holoprosencephaly 0.608 established (curated) no MR -> candidate analysis
lobar holoprosencephaly 0.608 established (curated) no MR -> candidate analysis
microform holoprosencephaly 0.608 established (curated) no MR -> candidate analysis
alobar holoprosencephaly 0.608 established (curated) no MR -> candidate analysis
septopreoptic holoprosencephaly 0.608 established (curated) no MR -> candidate analysis
semilobar holoprosencephaly 0.608 established (curated) no MR -> candidate analysis
Abnormality of the skeletal system 0.216 common-variant locus no MR -> candidate analysis

Of the 7 rows above, 7 have no MR estimate in this resource. Across all retrieved diseases for this gene: 0 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability 1 known modulators (Protein Cripto)
gnomAD constraint not available
GWAS Catalog no mapped SNPs
ClinVar no records
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance