CausalSentinel

Protein Dossier — TREML1 (Trem-like transcript 1 protein)

MR feasibility tier: A — Published pQTL-MR estimates exist for this protein (retrieved below - not computed here).

1. Published MR estimates (retrieved, not computed)

Outcome beta se p method nSNP cis/trans coloc
Vascular or heart problems diagnosed by doctor: Angina 0.229 0.0764 0.00269 Wald ratio 1 cis NA
Diastolic blood pressure automated reading -0.0459 0.0174 0.00838 Wald ratio 1 cis NA
Eye problems or disorders: Diabetes related eye disease 0.396 0.151 0.00861 Wald ratio 1 cis NA
Fractured bone site(s): Wrist 0.238 0.0968 0.0139 Wald ratio 1 cis NA
Breast cancer (Combined Oncoarray; iCOGS; GWAS meta analysis) -0.103 0.0427 0.0159 Wald ratio 1 cis NA
High grade serous ovarian cancer -0.249 0.108 0.0204 Wald ratio 1 cis NA
Red blood cell count 0.0341 0.0161 0.0342 Wald ratio 1 cis NA
Neo-extraversion -1.18 0.59 0.0447 Wald ratio 1 cis NA
Years of schooling -0.0474 0.0237 0.0455 Wald ratio 1 cis NA
Packed cell volume 0.248 0.124 0.0459 Wald ratio 1 cis NA
Body fat -0.0796 0.0412 0.0535 Wald ratio 1 cis NA
Diagnoses - main ICD10: K80 Cholelithiasis 0.189 0.0992 0.0562 Wald ratio 1 cis NA
…and 93 more outcomes (see JSON)              

2. pQTL instrument availability (Tier-B probe)

No prot-* pQTL GWAS dataset found for this protein (matched by UniProt accession and symbol).

3. GWAS Catalog results — traits with signal at this locus

4 association rows across 4 traits (3 genome-wide significant rows). Associations are loci, not causal claims; the mapped gene at a locus is not necessarily the effector gene.

Trait best p lead SNP n assoc study MR status
Triggering receptor expressed on myeloid cells 2 level in Ch 7e-30 rs73427270 1 GCST90234514 no MR -> candidate analysis
TREML2 protein levels 3e-16 rs114027364 1 GCST90470962 no MR -> candidate analysis
Circulating TREML2 levels 6e-14 rs547564061 1 GCST90859936 no MR -> candidate analysis
Alzheimer’s disease, proxy Alzheimer’s disease or related de 2e-6 rs116748189 1 GCST90654664 no MR -> candidate analysis

4. Phenome map — where this gene is a genetic locus, vs. where MR exists

Top diseases by Open Targets association (of 214 total). Associations are loci, not causal claims. The causal-status column is a four-state triage per pair: established (curated) = a curated clinical assertion exists (ClinGen/G2P/GEL/Orphanet/ClinVar — any validity level, MR adds little); exploratory rare-variant signal = ExWAS burden evidence without curation — a candidate NEW gene-disease relationship; common-variant locus = GWAS signal, classic pQTL-MR territory; multi-layer = burden+GWAS together, an allelic-series candidate (the strongest causal setup). Burden estimand is carrier-vs-noncarrier, not per-SD MR.

Disease genetic assoc. burden (ExWAS) causal status MR status
Parkinson disease 0.152 0.152 exploratory rare-variant signal no MR -> candidate analysis
Alzheimer disease 0.133 common-variant locus no MR -> candidate analysis
late-onset Alzheimers disease 0.084 common-variant locus no MR -> candidate analysis
neurodegenerative disease 0.084 common-variant locus no MR -> candidate analysis

Of the 4 rows above, 4 have no MR estimate in this resource. Across all retrieved diseases for this gene: 1 exploratory rare-variant signal(s), 0 multi-layer (allelic-series candidate) pair(s). Final triage still belongs to a statistical geneticist.

5. Downstream annotation (druggability & safety preview)

Layer Result
ChEMBL druggability not available — no ChEMBL target (undrugged)
gnomAD constraint pLI=3.6e-11, LOEUF=1.64 — LoF-tolerant
GWAS Catalog 68 unique SNPs / 136 rows
ClinVar 54 records; 0 pathogenic in sample of 30
PharmGKB/ClinPGx no annotations

Caveats declared by the tools

Sources

Provenance